doi: 10.1210/me.2011-0065 32 NeirijnckYCalvelPKilcoyneKRKhneFStvantIGriffethRJet al
These include decreased drug transport, increased cellular detoxification because of increased glutathione (GSH), changes in DNA repair involving increased nucleotide excision repair and/or loss of mismatch repair, increased tolerance of DNA adducts, and alterations in the apoptotic cell death pathway (Andrews and Howell, 1990
~ AlphaMD Most men on testosterone replacement therapy focus entirely on the hormone itself, assuming the prescription does all the work
activation of PI3K/AKT downstream pathway driving synaptogenesis and anti-apoptotic effects in hippocampus Regulatory status: not approved by the FDA or EMA
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The Safety of Glutathione for G6PD Deficiency The question of safety often arises because people with G6PD deficiency are told to avoid "oxidizing agents." Since glutathione is an antioxidant, it is the opposite of an oxidizer