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A clinical trial showed that vitamin E can significantly increase the risk of prostate cancer in men 109

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[28] [29] Consequently, an altered gene expression can possibly mediate impaired foetal growth and the programming of non-communicable diseases

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For instance, upregulating GPX4 in ferroptosis-sensitive chondrocytes, restoring antioxidant defenses in inflammatory synovial macrophages, or rebalancing iron export in subchondral osteoblasts may prevent tissue-specific degeneration

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