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In epithelial cells, E-cadherin inhibits ferroptosis by mediating the interaction of intracellular NF2 (also called merlin) and Hippo signaling pathway while antagonizing the signal axis mediated by E-cadherin promotes ferroptosis through YAP [72]

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[34] did not observe significant differences in high-intensity power parameters (peak and average power), in percentage of fatigue, and in total work (kJ), after 8 weeks of supplementation