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CCL20 mediates lipopolysaccharide induced liver injury and is a potential driver of inflammation and fibrosis in alcoholic hepatitis

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Goal: Inhibit NNMT to raise intracellular NAD+ and activate SIRT1, theorized to support fat metabolism and preserve lean mass effects reported in animal and cell studies, not established in humans [1] [5]

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720 Specifically, the potential mechanisms may involve that the suppression of GSK3 induced by CR to protect PGC-1 from intranuclear proteasomal degradation and the induction of SIRT1 by CR to enhance the transcriptional activity of PGC-1