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In a murine model of NASH, mice that received Emricasan treatment were protected from liver injury and fibrosis, suggesting that inhibition of hepatocytes apoptosis by Emricasan may be an attractive antifibrotic strategy in NASH

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Mitochondrial dysfunction further exacerbates oxidative stress by impairing ATP production and damaging mitochondrial DNA, leading to compromised sperm motility and energy deficits

In severe cases, despite the impracticality due to social, familial, and economic constraints, permanent relocation to a lower altitude is advisable

Bull Exp Biol Med 2003;135:4958