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However, cancer cells can escape cellular ferroptosis by activating or remodeling various signaling pathways, including oxidative stress pathways, thereby limiting the efficacy of ferroptosis-activating targeted therapy

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Therefore, selenium in the form of hydrogen selenide participates in the biosynthesis of individual selenoproteins [44, 58] which neutralize excess reactive oxygen species

Single-cell metabolic fingerprints discover a cluster of circulating tumor cells with distinct metastatic potential

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