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GHK-Cu drives collagen production through activation of transforming growth factor-beta (TGF-) signaling, the most potent endogenous stimulator of collagen synthesis in fibroblasts

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parenterally or perorally) consistently antagonizes the NSAIDs effect on lower esophageal sphincter and pyloric sphincter, thus counteracting both the early effects and the late effects induced by non-specific and specific NSAIDs