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found that GHK-Cu stimulated collagen synthesis by fibroblasts, noting that the presence of a GHK triplet in type I collagen meant that GHK may offer in situ healing effects [1, 6]

However, these resulting effects of NRF2 induction via CDDO treatment were to a dramatically lower degree compared to the effects of NRF2 induction upon GSH depletion in Gclc L-KO mice (Figs

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