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2010;341:c5702

Introduction Cardiovascular diseases (CVDs) are the leading cause of death worldwide and rising health care costs [1]

Nevertheless, several cellular studies have suggested the protective roles of chemical anti-oxidants, such as GSH, as well as drug metabolizing enzymes, such as NQO1 and UDP-glucuronosyltransferases (UGTs), against AQ-induced cytotoxicity (Tafazoli and OBrien, 2009

It is also conceivable that longer intervention with GSH may show further improvements in glycemic parameters, such as fasting glycemia

Advances in the modulation of ROS and transdermal administration for anti-psoriatic nanotherapies