GSH actively attends to oxidative free radicals and, in doing so, becomes oxidized and converts to the inactive GSSG
Experimental Gerontology, 138 , 111016
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Pathogen proliferation drives the secretion of a toxin cocktail (-toxin, perfringolysin O, and 2-toxin) that disrupts the abomasal mucosal barrier, which triggers necrosis, hemorrhage, and intramural gas accumulation and ultimately leads to severe structural degradation of the abomasal wall 70,71
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The key is acting on early symptoms like bleeding gums before the disease progresses to advanced stages