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BPC-157's activation of the eNOS pathway and resulting nitric oxide production contributes additional anti-inflammatory benefits, as NO can suppress leukocyte adhesion and migration while promoting the resolution of inflammatory lesions

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[4] In contrast, methionine limitation in the diet decreased insulin resistance in rodents and promoted adipose tissue lipolysis and fatty acid oxidation

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No significant differences between treatment groups were observed using the Chao1 richness estimate 42 , which accounts for ASV rareness in the richness measure (Supplementary Fig

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