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doi: 10.1210/jc.2015-2222 243 Blow PedersenIKnudsenNCarlASchomburgLKhrleJJrgensenTet al

G.et al (2013)

Moreover, these exosomes partially inhibit premature activation and depletion of primordial follicles by modulating the PI3K/AKT/mTOR signaling pathway, which is typically hyperactivated by chemotherapy, resulting in follicle burnout. Following treatment with hAEC exosomes, there is a reduction in the phosphorylation levels of critical proteins, including Akt and FoxO3a, which contributes to the preservation of ovarian reserve

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