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Further support comes from cross-sectional studies comparing PD and matched control subjects 13 , while others have investigated the role of UA in disease progression and severity, with findings suggesting an influence of low UA in each of these clinical factors 14,15

The notable increase in Cx32 level in hepatocytes of hepsin / mice, along with our previous work establishing hepsin as a regulator of the cellular abundance of hepatic GJ proteins (Hsu et al

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