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Nature: New Biology

Journal of Drugs in Dermatology (2016)

GHK-Cu acts as a genomic modulatorit can upregulate a broad array of genes related to tissue growth and downregulate those linked to inflammation and tissue breakdown [1]

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Therefore, the amount of non-hydrolyzed proteins that can be released from yeast cells could explain the lower differences observed between the amount of protein release from the biomass treated by PEF 1 compared with the amount released from biomass subjected to the two more intense PEF treatments