42 Further analysis of DNA damage signalling demonstrates the causal role of P53 in low-dose CSC-induced endothelial cell death by showing an upregulation in P53 protein expression and the cell death protective activity of the P53 knock down
We found that relative to Non-M-CS exposure, M-CS exposure induced greater increases in the above-mentioned lung inflammatory indices, as well as in the epithelial and lung expression of TRPM8, p-ERK, and p-JNK (Figures 16)
Lynch J, Lorenz L, Brueggemeyer JL, Lanzarotta A, Falconer TM, Wilson RA (2021) Simultaneous temperature measurements and aerosol collection during vaping for the analysis of 9-tetrahydrocannabinol and vitamin E acetate mixtures in ceramic coil style cartridges
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