Participation of cathepsin L on bone resorption
NO is an endogenously produced vasodilator and is largely implicated in hemostatic mechanisms [39] while, in tail amputation and heparin-/warfarin-administration models, both the NO-synthase (NOS)-blocker, L-NAME (thought to have a prothrombotic effect) [3] and NOS-substrate, L-arginine [presented with an antithrombotic effect) [3] were little investigated
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Hofstetter CP, Schwarz EJ, Hess D, Widenfalk J, El Manira A, Prockop DJ, et al
Folic acid fortification has substantially reduced neural tube defects at the population level, which is an undeniable public health success
Crescenze IM, Gupta P, Adams G et al: Advanced management of patients with ulcerative interstitial cystitis/bladder pain syndrome
