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Research indicates that in genioglossus myogenic stem cells, hypoxia leads to increased HIF-1 expression and the formation of HIF-1 heterodimers, subsequently upregulating BNIP3 expression

The precise regulation of ALS pathogenetic mechanisms is still elusive, despite the abundance of identified mechanisms mediating MN degeneration and death, including neuroinflammation, glutamate excitotoxicity, altered RNA metabolism, mitochondrial dysfunction, impaired cytoskeletal integrity, altered axonal transport dynamics, OS [71, 84, 85]

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