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For these reasons, several kinds of literature support the selective therapeutic targeting of the 7nAChR in individuals affected by ASD [22, 48, 166, 167]

J Neurochem (1999) 73(3):130917.10.1046/j.1471-4159.1999.0731309.x 12 PerierCTieuKGuganCCaspersenCJackson-LewisVCarelliVet alComplex I deficiency primes Bax-dependent neuronal apoptosis through mitochondrial oxidative damage

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