Pre-oligodendrocytes, which are more vulnerable than immature neurons or mature oligodendrocytes, are most affected (Haynes et al., 2003)
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Treatment of necrosis involves the administration of antibiotics, oxygen therapy, or removal of dead tissue
Studies of another thiol-containing antioxidant N -acetylcysteine (NAC), at its intrinsic pH, showed that it destabilizes the biofilm matrix by chelating divalent cations (e.g., Ca 2+ and Mg 2+ ), inhibition of bacterial extracellular polysaccharide production, modulation of bacterial cell physiology and the impact on its metabolism ( Our investigation of H 2 O 2 production by GSH during disruption and killing of MRAB biofilms produced interesting results (Figures 5CE)
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BPC-157 targets tissue repair and mucosal healing