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Conclusion: PQQ can regulate the pathogenesis of myocardial hypertrophy through the induction of YAP-related anti-ferroptotic activity, highlighting the potential value of PQQ as a novel therapeutic agent capable of slowing or preventing the progression of myocardial hypertrophy and thus delaying the onset of heart failure

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No single intervention will suffice

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Dosing Parameters in Clinical Settings Dose Range and Route Selection Preclinical dose-response studies demonstrate BPC-157 efficacy across an exceptionally wide dose range, from 10 nanograms per kilogram to 10 milligrams per kilogram, spanning six orders of magnitude

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